Endocrinology, acute medicine and critical care · FICTIONAL CASE

Acidosis with glucose 11: do not let the number hide DKA

Recognise SGLT2-associated euglycaemic ketoacidosis and continue insulin safely until ketosis resolves.

Pattern recognition and treatment-target discipline15 minutes0/3 decisions explored
PAUSE BEFORE USING

This is educational content, not a clinical protocol. Verify current guidance, use local pathways and seek appropriate senior or specialist help.

THE CASE

A fictional 48-year-old with type 2 diabetes taking dapagliflozin presents after vomiting and very limited intake. They are tachypnoeic and drowsy; glucose is 11.2 mmol/L, blood ketones 4.2 mmol/L, venous pH 7.18 and bicarbonate 10 mmol/L.

01 · INITIAL PRESENTATION

Listen for the assumption.

SHO

The glucose is only 11.2, so I ruled out diabetic ketoacidosis.

Registrar

The ketonaemia and metabolic acidosis define the danger. SGLT2 inhibitors can keep glucose only moderately raised while ketoacidosis develops.

SHO

I will stop dapagliflozin, start the local DKA pathway and use glucose-containing fluid as needed so insulin can continue safely to clear ketones.

WHERE IS THE CONSULTANT?

Choose an answer below and reveal the reasoning to see consultant feedback at every checkpoint. The full consultant-led synthesis follows after all three decisions.

Go to the first decision ↓
EVOLVING INFORMATION

The picture develops.

  1. Blood ketones 4.2 mmol/L
  2. Venous pH 7.18 and bicarbonate 10 mmol/L
  3. Dapagliflozin continued during vomiting and restricted intake
02 · DECISION CHECKPOINTS

Commit before you reveal.

After you select an option, reveal the model reasoning and the consultant’s feedback.

DECISION 1Which result should drive recognition?
DECISION 2Why may intravenous glucose be needed during treatment?
DECISION 3What is the safest SGLT2-inhibitor action?
03 · CONSULTANT-LED SYNTHESIS

Hear the senior team refine the plan.

The registrar tests the plan, the consultant synthesises the key principle, and the SHO confirms the safer next steps.

Registrar

Use the current JBDS/local DKA protocol, replace fluid and potassium appropriately, monitor ketones and pH, and look for precipitating illness.

Consultant

Do not improvise exact infusion rates outside the protocol. Involve the diabetes team and ensure basal insulin and transition decisions are actively reviewed.

SHO

I will hand over ketone and pH trajectories, insulin and glucose infusions, potassium, precipitant and the SGLT2-inhibitor plan.

CLINORA DECISION TRACE
01 · CUEKetonaemic acidosis with only moderate hyperglycaemia during SGLT2 therapy
02 · INTERPRETEuglycaemic diabetic ketoacidosis
03 · ACTStop the SGLT2 inhibitor and treat DKA to biochemical resolution
SAFETY CHECK

What must remain explicit.

  • Use the current local/JBDS adult DKA protocol.
  • Monitor glucose, blood ketones, pH, potassium and fluid status at protocol-defined intervals.
  • Seek senior/critical-care review for severe acidosis, altered consciousness or deterioration.
COMMON ERRORS

Where reasoning fails.

  • Excluding DKA because glucose is below 14 mmol/L
  • Stopping insulin before ketosis resolves
  • Restarting the SGLT2 inhibitor without specialist review
STRUCTURED HANDOVER
SGLT2-associated euglycaemic DKA with glucose [trend], ketones/pH/bicarbonate [trend], insulin/glucose/potassium [current therapy], fluid balance [status], precipitant [assessment] and medicine plan [details].
REFLECTIVE PAUSE

Does your acute clerking prompt ketone testing in an unwell person taking an SGLT2 inhibitor despite near-normal glucose?